Adipokines, Inflammation, and Metabolic Diseases
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A clear understanding of the pathophysiological processes in the adipose tissue and the interplay between obesity and chronic inflammation can lead to a better understanding of the development of comorbidities and may ensure future targets for the treatment of obesity.
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Obesity is an important condition affecting the quality of life of numerous patients and increasing their associated risk for multiple diseases, including tumors and immune-mediated disorders. Inflammation appears to play a major role in the development of obesity and represents a central point for the activity of cellular and humoral components in the adipose tissue. Macrophages play a key role as the main cellular component of the adipose tissue regulating the chronic inflammation and modulating the secretion and differentiation of various pro- and anti-inflammatory cytokines. Inflammation also involves a series of signaling pathways that might represent the focus for new therapies and interventions. Weight loss is essential in decreasing cardiometabolic risks and the degree of associated inflammation; however, the latter can persist for long after the excess weight is lost, and can involve changes in macrophage phenotypes that can ensure the metabolic adjustment. A clear understanding of the pathophysiological processes in the adipose tissue and the interplay between obesity and chronic inflammation can lead to a better understanding of the development of comorbidities and may ensure future targets for the treatment of obesity.
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@article{SvulescuFiedler2024Interplay,
title = {The Interplay between Obesity and Inflammation},
author = {Ilinca Săvulescu-Fiedler and R. Mihalcea and Şerban Dragosloveanu and Cristian Scheau and Radu Octavian Baz and Ana Căruntu and Andreea-Elena Scheau and Constantin Căruntu and Șerban Benea},
journal = {Life},
year = {2024},
doi = {10.3390/life14070856},
url = {https://doi.org/10.3390/life14070856}
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